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Prolonged Stimulation Of Growth Hormone GH And Insulin-like
Growth Factor I Secretion By CJC-1295, A Long-acting Analog
Of GH-releasing Hormone, In Healthy Adults

Account

The account section provides a quick overview of the user profile associated with this publication entry.
It includes basic identification details such as the username, institutional affiliation,
and access privileges granted to the researcher or reader.

For those who are registered within the database, this section also lists the number of saved
references, collections created, and any ongoing projects that incorporate the
article.

Save citation to file

This feature allows users to export a bibliographic record in several standard formats (e.g.,
RIS, BibTeX, EndNote XML). By clicking the "Save citation to file" button, the user can download a text file containing all metadata fields—title, authors, abstract,
publication details, and identifiers such as PMID and DOI.
The exported file is ready for import into reference management software or for archival purposes.

Email citation

The "Email citation" function composes an email with the article’s
citation embedded in the message body. Users can quickly share a
complete reference with colleagues or collaborators without manually copying the information. The email includes all
essential fields—authors, title, journal, volume, issue, pages, year,
and DOI—to ensure proper attribution.

Add to Collections

Researchers often organize literature into thematic collections for ease of retrieval.
By selecting "Add to Collections," a user can place this article into one or multiple pre‑defined folders such as "Growth Hormone Studies,"
"Endocrine Therapy Trials," or "Clinical Pharmacology." This categorization supports
personalized research workflows and aids in generating topic-specific bibliographies.

Add to My Bibliography

The "Add to My Bibliography" option inserts the citation directly
into a user’s personal bibliography database.
The entry is stored with all relevant metadata, allowing future access via search or cross‑reference functions.
It also integrates seamlessly with external
tools like Zotero or Mendeley if the user has linked their account.

Your saved search

When a user creates a custom query—filtering by keywords, publication date, or subject area—the resulting list of articles is stored as a "saved search." This feature automatically updates whenever
new literature matching those criteria becomes
available. The article in question appears in the results if it satisfies the saved search parameters.

Create a file for external citation management software

This function generates a ready‑to‑import file that
can be loaded into third‑party reference managers
such as EndNote, RefWorks, or JabRef. The exported data includes
all necessary fields—author list, title, abstract, MeSH terms, and PubMed identifiers—to populate the target
program’s database without manual entry.

Your RSS Feed

The RSS feed link delivers updates on new publications related to the article’s subject area.

By subscribing, users receive notifications whenever a new
study on growth hormone analogs or insulin‑like growth factor research is indexed in the database,
ensuring continuous awareness of emerging evidence.

Full text links

This section aggregates all available full‑text sources for the article.
It lists open access PDFs, publisher-hosted HTML versions, institutional repositories, and other repositories such as PubMed Central.
Each link includes a brief description (e.g., "Open Access PDF"

and the date of the most recent update.

Actions

The "Actions" menu provides quick tools for
interacting with the record: downloading PDFs, exporting citations,
sharing via social media or email, and printing the reference card.

It also offers options to create alerts, request full‑text from interlibrary loan services, and view citation metrics like impact factor and usage statistics.

Page navigation

The page navigation bar displays pagination controls for browsing
through multiple pages of search results or supplementary material associated with
the article. Users can jump directly to the first, last, next, or previous pages,
as well as enter a specific page number.

Affiliation

This field lists the institutional affiliations of all authors at the time
of publication. It may include universities, research institutes, and clinical centers.
For multi‑institutional studies, each affiliation is enumerated with corresponding superscript markers linked to the author list.

Authors

The authorship section enumerates every contributor in order
of appearance. Each name is accompanied by a brief note on their role—e.g.,
"Principal Investigator," "Data Analyst," or "Corresponding Author."
Where applicable, ORCID identifiers are displayed for unique researcher identification.

Abstract

The abstract provides a concise summary of the study’s
objectives, methodology, key findings, and conclusions. It highlights that CJC‑1295, a long‑acting analog of growth hormone‑releasing
hormone, was administered to healthy adults to assess
its impact on prolonged secretion of growth hormone (GH) and insulin‑like
growth factor I (IGF‑I). The results indicated sustained elevations in both hormones compared with placebo
controls, suggesting therapeutic potential for conditions requiring enhanced anabolic activity.

Publication types

The article is classified under several publication types:
Clinical Trial, Review Article, and Pharmacology Study.
These categories help users filter literature by study design or content focus when searching the database.

MeSH terms

Medical Subject Headings (MeSH) associated with this publication include:

Growth Hormone

Insulin‑Like Growth Factors

Peptides

Hormonal Therapy

Clinical Trials as Topic

These controlled vocabulary terms facilitate precise
indexing and retrieval in biomedical searches.

Substances

The primary substance investigated is CJC‑1295, a synthetic peptide analog of growth hormone‑releasing hormone (GHRH).

The article also references endogenous GH and IGF‑I, describing their biochemical pathways and physiological roles in metabolism and tissue repair.

LinkOut – more resources

LinkOut aggregates external resources linked to the record.
It provides direct access to related databases such as ClinicalTrials.gov for trial registration, FDA
approval status, drug safety profiles on Drugs@FDA, and educational materials on endocrine physiology from reputable institutions.

Full Text Sources

The full‑text sources section lists all platforms where the complete article is available.
This includes:

Publisher’s website (PDF/HTML)

PubMed Central

Institutional repository

Open access archives

Each source link opens in a new browser tab, enabling immediate reading or download of the research document.

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